By Ramana S. Moorthy MD
Starts off with an in-depth evaluation of immunemedicated eye sickness, summarizing simple immunologic ideas, ocular immune responses and detailed issues in ocular immunology. Discusses the scientific method of uveitis and experiences noninfectious (autoimmune) and infectious varieties of uveitis, with an increased part on viral uveitis and new fabric on infectious and noninfectious scleritis. stronger detection of infectious brokers by means of immunologic and genetic tools and new biologic therapeutics are distinctive. additionally covers endophthalmitis, masquerade syndromes, issues of uveitis and ocular elements of AIDS. encompasses a variety of new colour photos. significant revision 2011-2012
Read Online or Download 2011-2012 Basic and Clinical Science Course, Section 9: Intraocular Inflammation and Uveitis (Basic & Clinical Science Course) PDF
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Additional resources for 2011-2012 Basic and Clinical Science Course, Section 9: Intraocular Inflammation and Uveitis (Basic & Clinical Science Course)
Afferent lymphatic channels are veinlike structures that drain extracellular fluid (ie. lymph) from a site into a regional node. Lymphatics serve 2 major purposes: to convey immune cells and to carry whole antigen from the site of inoculation to a lymph node. Antigen-presenting cells APCs are specialized cells that bind and phagocytize antigen at a site. Following ingestion of antigen. APCs migrate to lymph nodes. where they process the antigen. typically by intracellular proteolysis to short amino acid chains of7 to II amino acids.
Over the next few days, as T and B lymphocytes are primed, the hilar nodes become enlarged because of th e increased number of dividing T and B lymphocytes as well as by the genera lized increased traffi cki ng of other lymphocytes through the nodes. The effector phase begins when the primed T lymphocytes recirculate and enter the infected lung. The T lymphocytes interact with t he macrophageingested bacteria, and cytokines are released that activate neighboring macrophages to fu se into giant cells, forming caseating granulomas.
These molecules and cells are conveyed back to the original site. where an effector response 33 34 • Intraocular Inflammation and Uveitis CLINICAL EXAMPLES 2-1 Primary response to poison ivy toxin The first contact between the poison ivy resin urushiol and the epidermis triggers the immunologic mechanisms of poison ivy dermatitis. The afferent phase of this primary response begins when the toxin permeates into the epidermis, where it binds to extracellu· lar proteins, forming a protein-toxin conjugate technically called a hapten.
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